stealth rnai sirna oligonucleotides negative control med gc scramble (Thermo Fisher)
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Stealth Rnai Sirna Oligonucleotides Negative Control Med Gc Scramble, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/stealth+sirna+negative+control+(scramble)/stealth+rnai+sirna+oligonucleotides+negative+control+med+gc+scramble/pm34485842-80-0-4
Average 90 stars, based on 1 article reviews
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other:Article Title: Intranasal CRMP2-Ubc9 Inhibitor Regulates Na V 1.7 to Alleviate Trigeminal Neuropathic Pain Article Snippet: For the corresponding set of experiments, trigeminal ganglion neurons (TGs) were transfected with EGFP and Invitrogen Article Title: BLID is a drug-responsive target of FOXO3a and multi-omics analysis reveals survival mechanisms and therapeutic vulnerabilities in BLID-deficient breast cancer cells Article Snippet: In addition, Stealth siRNA negative control (scramble) with the same supplier's proprietary sequence designed to minimize sequence homology to any known Article Title: Myosin Va interacts with the exosomal protein spermine synthase Article Snippet: Supplementary method Western blotting MCF-7 cells were plated and transfected in 6-well plates using DharmaFECT 1 Transfection Reagent (GE Healthcare) according to the manufacturer’s instructions, with the Stealth RNAiTM siRNA targeting MYO5A and the Small Interfering RNA:Article Title: PGC-1α and PGC-1β increase CrT expression and creatine uptake in myotubes via ERRα. Article Snippet: .. Myotubeswere transfectedwith 60 pmol pre-designed Stealth RNAi TM short interfering RNA (siRNA) for the CrT (Slc6A8) (cat no. RSS328932 and RSS328934) or the Article Title: The Role of MKP-5 in Adipocyte-Macrophage Interactions during Obesity Article Snippet: Human 293A cells from the ATCC were cultured using DMEM containing 10% FBS and penicillin/streptomycin. .. RNA Interference Small interfering RNA (siRNA) oligoribonucleotides targeting MKP-5 (siMKP5; Stealth RNAi TM siRNA, Cat. ‣8927232) and Article Title: The protective role of the MKP-5-JNK/P38 pathway in glucolipotoxicity-induced islet β-cell dysfunction and apoptosis. Article Snippet: Hyperglycemia and hyperlipidemia (glycolipotoxicity)-triggered islet β-cell dysfunction is known to drive the progression of obesity-related type 2 diabetes, however the underlying mechanisms have not been clearly elucidated.. The current study aimed to investigate the role of mitogen-activated protein kinase phosphatase 5 (MKP-5) in islet cells under glucolipotoxic conditions.. Using gene overexpression and knockdown approaches, we demonstrated that MKP-5 could alleviate glucolipotoxicity-induced apoptosis via the endoplasmic reticulum (ER) stress and mitochondrial apoptosis pathways owing to the altered regulation of caspase family members and ER stress-related molecules in MIN6 and primary islet cells. Negative Control:Article Title: PGC-1α and PGC-1β increase CrT expression and creatine uptake in myotubes via ERRα. Article Snippet: .. Myotubeswere transfectedwith 60 pmol pre-designed Stealth RNAi TM short interfering RNA (siRNA) for the CrT (Slc6A8) (cat no. RSS328932 and RSS328934) or the Article Title: The Role of MKP-5 in Adipocyte-Macrophage Interactions during Obesity Article Snippet: Human 293A cells from the ATCC were cultured using DMEM containing 10% FBS and penicillin/streptomycin. .. RNA Interference Small interfering RNA (siRNA) oligoribonucleotides targeting MKP-5 (siMKP5; Stealth RNAi TM siRNA, Cat. ‣8927232) and Article Title: The protective role of the MKP-5-JNK/P38 pathway in glucolipotoxicity-induced islet β-cell dysfunction and apoptosis. Article Snippet: Hyperglycemia and hyperlipidemia (glycolipotoxicity)-triggered islet β-cell dysfunction is known to drive the progression of obesity-related type 2 diabetes, however the underlying mechanisms have not been clearly elucidated.. The current study aimed to investigate the role of mitogen-activated protein kinase phosphatase 5 (MKP-5) in islet cells under glucolipotoxic conditions.. Using gene overexpression and knockdown approaches, we demonstrated that MKP-5 could alleviate glucolipotoxicity-induced apoptosis via the endoplasmic reticulum (ER) stress and mitochondrial apoptosis pathways owing to the altered regulation of caspase family members and ER stress-related molecules in MIN6 and primary islet cells. Article Title: Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons Article Snippet: The supernatant was discarded, and the pellet was resuspended in complete DMEM media containing 1 % penicillin/streptomycin sulfate (10,000 μL −1 , stock), 30 ng mL −1 of nerve growth factor and 10 % fetal bovine serum (Hyclone). .. Collected nodose ganglion neurons were then transfected with Numb plasmid (pCMV-FLAG-Numb) , Invitrogen Synthesized:Article Title: The Role of MKP-5 in Adipocyte-Macrophage Interactions during Obesity Article Snippet: Human 293A cells from the ATCC were cultured using DMEM containing 10% FBS and penicillin/streptomycin. .. RNA Interference Small interfering RNA (siRNA) oligoribonucleotides targeting MKP-5 (siMKP5; Stealth RNAi TM siRNA, Cat. ‣8927232) and Article Title: The protective role of the MKP-5-JNK/P38 pathway in glucolipotoxicity-induced islet β-cell dysfunction and apoptosis. Article Snippet: Hyperglycemia and hyperlipidemia (glycolipotoxicity)-triggered islet β-cell dysfunction is known to drive the progression of obesity-related type 2 diabetes, however the underlying mechanisms have not been clearly elucidated.. The current study aimed to investigate the role of mitogen-activated protein kinase phosphatase 5 (MKP-5) in islet cells under glucolipotoxic conditions.. Using gene overexpression and knockdown approaches, we demonstrated that MKP-5 could alleviate glucolipotoxicity-induced apoptosis via the endoplasmic reticulum (ER) stress and mitochondrial apoptosis pathways owing to the altered regulation of caspase family members and ER stress-related molecules in MIN6 and primary islet cells. Transfection:Article Title: The protective role of the MKP-5-JNK/P38 pathway in glucolipotoxicity-induced islet β-cell dysfunction and apoptosis. Article Snippet: Hyperglycemia and hyperlipidemia (glycolipotoxicity)-triggered islet β-cell dysfunction is known to drive the progression of obesity-related type 2 diabetes, however the underlying mechanisms have not been clearly elucidated.. The current study aimed to investigate the role of mitogen-activated protein kinase phosphatase 5 (MKP-5) in islet cells under glucolipotoxic conditions.. Using gene overexpression and knockdown approaches, we demonstrated that MKP-5 could alleviate glucolipotoxicity-induced apoptosis via the endoplasmic reticulum (ER) stress and mitochondrial apoptosis pathways owing to the altered regulation of caspase family members and ER stress-related molecules in MIN6 and primary islet cells. Article Title: Insulin-independent stimulation of skeletal muscle glucose uptake by low-dose abscisic acid via AMPK activation Article Snippet: Transient transfection of L6 cells was performed using the Nucleofector System (Amaxa GmbH, Cologne, Germany). .. L6 (2 × 10 6 cells) were transfected using the Nucleofector program X-005 and R Kit with 2 μM rat LANCL2-targeting Stealth duplex short interference RNA (siRNA-L2, NM_001014187_stealth_208) or with 2 μM Article Title: Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons Article Snippet: The supernatant was discarded, and the pellet was resuspended in complete DMEM media containing 1 % penicillin/streptomycin sulfate (10,000 μL −1 , stock), 30 ng mL −1 of nerve growth factor and 10 % fetal bovine serum (Hyclone). .. Collected nodose ganglion neurons were then transfected with Numb plasmid (pCMV-FLAG-Numb) , Invitrogen Construct:Article Title: The protective role of the MKP-5-JNK/P38 pathway in glucolipotoxicity-induced islet β-cell dysfunction and apoptosis. Article Snippet: Hyperglycemia and hyperlipidemia (glycolipotoxicity)-triggered islet β-cell dysfunction is known to drive the progression of obesity-related type 2 diabetes, however the underlying mechanisms have not been clearly elucidated.. The current study aimed to investigate the role of mitogen-activated protein kinase phosphatase 5 (MKP-5) in islet cells under glucolipotoxic conditions.. Using gene overexpression and knockdown approaches, we demonstrated that MKP-5 could alleviate glucolipotoxicity-induced apoptosis via the endoplasmic reticulum (ER) stress and mitochondrial apoptosis pathways owing to the altered regulation of caspase family members and ER stress-related molecules in MIN6 and primary islet cells. Control:Article Title: Insulin-independent stimulation of skeletal muscle glucose uptake by low-dose abscisic acid via AMPK activation Article Snippet: Transient transfection of L6 cells was performed using the Nucleofector System (Amaxa GmbH, Cologne, Germany). .. L6 (2 × 10 6 cells) were transfected using the Nucleofector program X-005 and R Kit with 2 μM rat LANCL2-targeting Stealth duplex short interference RNA (siRNA-L2, NM_001014187_stealth_208) or with 2 μM Plasmid Preparation:Article Title: Cell specific regulation of NaV1.7 activity and trafficking in rat nodose ganglia neurons Article Snippet: The supernatant was discarded, and the pellet was resuspended in complete DMEM media containing 1 % penicillin/streptomycin sulfate (10,000 μL −1 , stock), 30 ng mL −1 of nerve growth factor and 10 % fetal bovine serum (Hyclone). .. Collected nodose ganglion neurons were then transfected with Numb plasmid (pCMV-FLAG-Numb) , Invitrogen |

